A small protein previously associated with cellular dysfunction and death in fact serves a critical function in repairing breaks in DNA, according to new research. The study is the first to demonstrate the role that alpha-synuclein plays in forestalling the demise of neurons in brain diseases such as Parkinson’s. The findings suggest that it may be possible to design new therapies to replace alpha-synuclein’s function or boost it in people with Parkinson’s disease and other neurodegenerative disorders.
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Source: ScienceDaily