Researchers from the University of Seville and the Andalusian center of Molecular Biology and Regenerative Medicine (CABIMER), have discovered a new mechanism that makes it possible to understand premature aging in cells with asymmetrical cell division, as is the case with mother cells. Understanding this mechanism could be useful for studying and anticipating the development of aging-related diseases such as cancer and neurodegenerative processes. The research was published in Nature Cell Biology.
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Source: Phys.org