Traditionally, bacterial toxins have been seen as killers of target cells. But is there more than meets the eye? UmeƄ University Professor Teresa Frisan and her team have discovered that toxin-host interactions are more complex and that bacterial toxins can act as modulators of the host immune response to avoid bacteria detection and long-term infection. The study has been published in the journal Cell Reports.
Click here for original story, A bacterial toxin that acts as a mediator rather than a killer
Source: Phys.org